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Conolidine’s analgesic results stem from its interaction with non-opioid pain pathways. Contrary to opioids, which bind to µ-opioid receptors during the central anxious technique, conolidine modulates alternate molecular targets. A Science Innovations analyze identified that conolidine interacts Using the atypical chemokine receptor ACKR3/CXCR7,
Some have now been studied as prospect prescription drugs, but they are opioids, belonging to the exact same course of compounds as morphine and hydrocone.Additionally, opioid-induced hyperalgesia—a problem the place prolonged opioid use heightens pain sensitivity—hasn't been observed in preclinical assessments of conolidine. Contrary to NSAIDs
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